Coverjam for iOS creates Instagram and Flickr-powered slideshows of your favorite bands

Earlier this week, we wrote about British band the Vaccines using Instagram to crowdsource the creation of its new music video.

Today, we bring you Coverjam, a new iOS app that automatically taps into tagged images on Instagram and Flickr to create slideshows which run in tandem when an artist?s music is playing on your portable device.

When the app is opened, Coverjam loads the music directly from the iOS device, from where users can select songs, albums or playlists. If you prefer, you can select music directly from the device?s built-in music player and the slideshow will simply play over the top.

A fresh set of slides launch each time a new song starts, or if you want to load a new batch of photos manually, simply hit the refresh button. You can also ban photos that you don?t like so they?re not shown again within the slideshow.

As with any app these days, Coverjam has a social feature built in, so you can share photos that you like across Facebook, Twitter and eMail.

Iain Barclay, Chief Product Officer at CoverJam?s developer Electric Pocket, said:

?Coverjam is the perfect way for an artist?s fans to see the latest and greatest images of the acts they love. Whether it?s the latest tour photos, new photo-shoots, or art created by other fans, the photos in Coverjam are sure to delight the most avid music lovers.?

The free version of Coverjam includes advertising and is available for iPhone and iPod Touch devices, however it will run in 2x mode on iPads. Coverjam Pro has no ads, and offers a full-screen functionality on iPads, iPhones and iPod touches, at a cost of US$0.99.

Check out the official Coverjam promo video for the app here:

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Source: http://thenextweb.com/apps/2011/07/20/coverjam-for-ios-creates-instagram-and-flickr-powered-slideshows-of-your-favorite-bands/

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Alzheimer's Detected 20 Years before Symptoms Show

For the first time, scientists have been able to detect signs of Alzheimer's disease 10 to 20 years before the onset of dementia. The study, presented Wednesday at the Alzheimer's Association International Conference in Paris, focused on people with rare, inherited forms of the disease who develop it relatively young, with symptoms beginning in the patients' 30s, 40s, and 50s. Researchers say the results will help them test drugs that could prevent or slow the progression of the disease, not only in these groups, but also in people with the more common, late-onset variety.

It's proven to be extremely difficult to develop effective treatments for Alzheimer's. One possible reason is that new drugs are tested too late in the progression of the disease; by the time memory problems become evident, extensive brain damage has already taken place. In the last few years, scientists have renewed efforts to find ways to detect the disease earlier, including brain imaging, blood tests, and tests of cerebral spinal fluid (CSF).

Randall Bateman, a neurologist at Washington University School of Medicine, and collaborators have been searching for such predictors by studying families with inherited versions of Alzheimer's. In these cases, a single copy of the mutated gene guarantees that the carrier will develop the disease. Scientists can estimate the age of the disease's onset based on the affected parent; that allows them to look for physiological changes decades before memory impairments become evident. The research is part of a large, multicountry study called DIAN (for Dominantly Inherited Alzheimer's Network).

While this type of Alzheimer's is rare, accounting for about 1 percent of cases, both inherited and more common "sporadic" forms of the disease are characterized by excessive buildup of amyloid protein in the brain. (The mutations that cause early-onset Alzheimer's are different from those that increase the risk of developing the disease.)

"We can see changes over time, which allows us to estimate the order and magnitude of changes that occur leading up to Alzheimer's," says Bateman. "This is a way to tell us potentially how long of a window we have to treat it, and how we can use these markers in sporadic disease if the timing is the same."

Families in the DIAN study have mutations in three different genes, all of which are linked to the production or processing of amyloid. Researchers found that family members who carried the disease gene showed an increase amyloid beta protein in the blood and CSF about 30 years prior to the expected onset of the disease compared to noncarrier family members. Within 10 to 15 years of the expected onset, that high level dropped to lower than noncarriers, while levels of tau, another protein associated with the disease, increased. The results come from the analysis of 150 family members with no outward symptoms and an average age of 37.

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How to find that culprit tab or extension slowing down Google Chrome

Over the last few months, we?ve had a number of questions from people wondering how to check which tab or extension is causing Chrome to slow down.

If you?re a Chrome user you?ve undoubtedly had moments were you?ve tried to figure this out yourself. For the most part, Chrome?s pretty good: in this particular job I have countless tabs open all the time and it often doesn?t bat an eye. But some pages with too much Flash, JavaScript or what have you chew up the CPU or memory more than all the others combined and it seems more noticeable in Chrome than in other browsers.

The good news is you don?t have to install yet another extension that?ll contribute to resource consumption even more. Chrome has the tools you need built right in.

The Google Chrome Task Manager is a feature as old as Chrome itself, but it often goes forgotten by the world?s Chrome user base. You can access it through the Window > Task Manager and sort tabs and plugins by their memory, CPU and network usage and end processes right there.

Next time you?re trying to figure out which tab is the culprit, cut out the guess work with this handy tip.

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Source: http://thenextweb.com/google/2011/07/20/how-to-find-that-culprit-tab-or-extension-slowing-down-google-chrome/

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Cleaner Coal Faces an Uncertain Future

American Electric Power's recent decision to scrap plans to capture and sequester carbon dioxide at a West Virginia power station is just the latest in a string of cancellations of carbon capture and storage (CCS) projects. The moves have dimmed the prospects for carbon-free power generation from coal. However, a handful of CCS projects are moving forward?including one in Mississippi that broke ground in December?so it might be too early to completely write the technology off.

The U.S. Department of Energy's goal is to start five to 10 large CCS projects within the next five years. The DOE believes those projects could drive down the cost of CCS, which currently boosts generation costs by at least 44 percent?but the incentives it's offering have clearly not been sufficient to entice utilities. Low natural-gas prices have eroded coal's cost advantage, while a national policy to penalize carbon-dioxide emissions has yet to materialize. As a result, utilities have been unwilling to pursue CCS, even with the DOE footing half the bill. "The federal incentives offered to move the technology forward just aren't working," says Kurt Waltzer, a carbon storage expert with the Clean Air Task Force, a nonprofit environmental consulting firm based in Boston.

For example, Columbus, Ohio-based AEP walked away from a $334 million federal grant to cover half of its proposed CCS installation. The plan was to capture at least 90 percent of the carbon dioxide from a portion of the flue gases at its 1,300-megawatt power plant in New Haven, West Virginia. The 1.5 million tons per year of captured carbon dioxide was to be permanently stored in geologic formations below the plant. But expected supports did not come through. The U.S. Senate rejected a cap-and-trade bill last year (AEP supported the legislation), while Virginia and West Virginia's public utility commissions refused to pass along all of AEP's costs to ratepayers.

Basin Electric cited cost as a primary factor in its December decision to walk away from a similar CCS retrofit. The Bismarck, North Dakota-based rural electricity cooperative had secured $100 million in DOE funding for a $287 million project to capture a million tons of carbon dioxide annually at its coal-fired power station in Antelope Valley, North Dakota.

Utilities moving forward with CCS projects are closing the gap by selling their carbon dioxide to enhanced oil recovery operations, which inject carbon dioxide into oil wells to help push more oil to the surface. Such operations currently provide about 5 percent of U.S. domestic oil production, and could ultimately double U.S. oil reserves, according to a report last year by MIT's Energy Initiative.

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Google releases Google app for iPhone

Google has released the official Google+ app for the Apple iPhone. You can download it now directly from the Apple App Store.

Make sure that the version you grab is 1.0.1.1809 or later. Google mistakenly released a previous test version of the app which didn't have the stability and fixes that the latest version has. To check the version number, click on the gear icon on the top left of your app's homescreen and look right above the Help button.

Also, it's important to note that this app is just for the iPhone, not iOS in general. Google says that iOS5 is unsupported at this point and that iPad device support is coming soon. The search giant did not give details on timing for either though.

Google's official description of the app is as follows:

Google+ for mobile makes sharing the right things with the right people a lot simpler. Huddle lets you send super-fast messages to the people you care about most. And no matter where you are, the stream lets you stay in the loop about what your friends are sharing and where they?re checking in.

Google released Google+ last month in a clear attempt to compete in the social space with Facebook and Twitter. There are five basic components to Google+: Circles (for managing friends), Sparks (subscribe to specific interests for a constantly updated feed), Hangouts (video conversations with up to 10 friends at once), Instant Uploads (automatically uploads photos snapped with your mobile device), and Huddle (group chatting among Google+ peers).

Google+ has had an Android app from day one, but the iPhone version had to be approved by Apple first. Now that it has, Google+ is available on the two biggest mobile operating systems in the US. I wonder how long it will take for Google to release a Google+ for the rest of the mobile platforms.

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Source: http://www.techspot.com/news/44758-google-releases-google-app-for-iphone.html

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Stem Cells Help Irradiated Mice Grow New Brain Cells

Radiation treatment for brain cancer can be lifesaving, but it can come at a terrible cost. The radiation that kills cancer cells also kills brain cells, destroying memories, impairing intelligence, and causing confusion.

Charles Limoli and colleagues at the University of California, Irvine, have shown that stem cells could help reverse some of this damage. In a new paper in the journal Cancer Research, Limoli shows that it's possible to cause new brain cells to grow by injecting human neural stem cells into the brains of mice whose cognitive abilities had been damaged by radiation. The mice regained lost skills after the stem-cell treatment.

Stem cells have long been used to repair the damage caused by cancer treatment. Bone-marrow transplants for leukemia rely on stem cells to replenish blood cells, for instance. But Limoli says his team is the only one using neural stem cells to treat symptoms in the brain.

Several peers praised his work, calling it an important proof of the idea that human stem cells can repair neuronal damage.

"The results are very promising," says Howard B. Lieberman, professor of radiation oncology and environmental health sciences at Columbia University. "If the findings continue to be as positive as what's published in this paper, I would assume Dr. Limoli will take great effort to try to move it into the clinic as quickly as possible."

Limoli's team irradiated three groups of mice, later treating two of them with human neural stem cells. The third, a control group, received a sham surgery, but no cells were implanted. One month after the damage, 23 percent of implanted stem cells were active in the brains of the first group of mice. After four months, 12 percent were still active in the second group. Using cellular labeling, Limoli's team also showed that tens of thousands of new neurons and astrocyte cells had grown in the brains of the treated mice. The treated mice performed better than the untreated ones on cognitive tests, and recovered their preradiation abilities.

Protein activity in the treated mice suggests that the implanted stem cells are integrating into the brain, Limoli says, replacing cells that have been lost or damaged.

Both Limoli and Lieberman say the treatment could also be effective against "chemo brain," a side effect often reported by breast cancer patients who say their ability to focus and think clearly has been impaired by chemotherapy.

Rob Coppes, a radiation and stem-cell biologist at the University Medical Center Groningen, in the Netherlands, says he would next like to see Limoli test how long the benefits of the stem cells last. He also hopes Limoli will repeat his experiments using induced pluripotent stem cells (iPS cells), adult stem cells that have been converted back to an embryonic-like state. These would likely be the cells that doctors would use in patients. Ideally they'd be taken from the patients themselves to avoid an immune rejection.

It will be important to show that mice?and later, humans in a trial?don't reject these cells, and also that the stem cells don't trigger new cancers, says Coppes, who employs stem cells in his own work, which involves regenerating salivary glands.

Limoli plans to carry out further work involving human neuronal stem cells and iPS cells. He also wants to figure out the optimal time to transplant these stem cells into the brain.

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Gaming 29 - The Post-Pub Podcast

Gaming 29 - The Post-Pub Podcast

Posted on 17th Jul 2011 at 08:23 by Podcast with 9 comments

Custom PC veteran Phil Hartup and PC Pro's Mike Jennings join Joe and Paul for a late-night, post-pint rant. This episode of the podcast, perhaps because it's sponsored by alcohol, stumbles along with vague coherency through topics such as BioShock Infinite and Just Cause 2.

Mass Effect 2 is obligatorily drawn into the discussion too, as is tradition.

Boozy fumes aren't enough to stop us tackling the thorny issues, however - Phil explains why he expects Battlefield 3 will be a shoddy console port, while Joe shoots down the defence that 64-player multiplayer is something to be proud of.

*hic*


On top of that, Phil brings us a report on how APB: Reloaded is faring after being brought back from the dead, while Joe orates further on his favourite topic of the moment; Frozen Synapse.

As always, we've also got our weekly competition, which this time gives you a chance to win yourself a copy of Assassin's Creed: Brotherhood on the PC and Raving Rabbids on the Nintendo 3DS. You can also find out who won the last competition and bagged themselves a Roccat Vire Gaming Headset.

As ever, the bit-tech hardware podcast features music by Brad Sucks, and was recorded on Shure microphones. You can download the podcast direct, listen in-browser or subscribe through iTunes using the links below. Also, be sure to let us know your thoughts about the discussion in the forums.

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Source: http://feedproxy.google.com/~r/bit-tech/blog/~3/-4ayxHYG6jU/

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Google warns users of widespread Windows-only malware infection

Google is warning users who appear to be infected with a widespread malware infection when they use Google?s search product, the company said today.

Google engineers noticed suspicious search data recently and worked with IT security at affected companies to find the cause: malware that re-routes traffic to Google and other sites through a proxy. To what end the infection does this for is unknown.

Google does not have the ability to detect the presence of viruses on user?s computers, simply the suspicious network activity caused by the malware.

Google says it hopes to combat the spread of the infection by making sure users are aware of it and can update their anti-virus software and catch it. If you are infected, you?ll know ? the company is using a big, yellow warning that you can?t miss.

Google?s Matt Cutts says:

This is important: Google has been able to detect a large number of computers infected with a specific piece of malware. If you go to Google and do a search (any word will do) right now, check to see whether you get a ?Your computer appears to be infected? warning at the top of the search results. If you see the message, you need to clean up the infection from your machine.

He also notes that the infection only affects machines running Windows.

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Source: http://thenextweb.com/google/2011/07/20/google-warns-users-of-widespread-malware-infection/

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Hardware 25 - What, no Tanks?

Hardware 25 - What, no Tanks?

Posted on 8th Jul 2011 at 14:20 by Podcast with 11 comments

It's been a while since our last podcast so James, Paul, Antony and Harry had plenty to talk about when they took their seats in the studio this week.

First on the agenda was James and Paul's trip to Computex in Taiwan, where they got to see what the industry had planned for the next six months. Certain things were of particular interest to us though such as the LGA2011 boards being shown at the exhibition.

The other big slice of news that's hit since our last podcast is the launch of AMD's new desktop Lynx processors. The APUs (as AMD calls them) are potentially interesting for those looking for a low cost rig that's also capable of gaming.

Finally, we sneak in a little discussion about Intel's new 50-core maths co-processor card and attempt to answer a reader question about thermal compound.

As always, we've also set up our weekly competition, the lucky winner of which will walk away with a brand new Corsair VX550W PSU.

Hardware 25 - What, no Tanks?

As ever, the bit-tech hardware podcast features music by Brad Sucks, and was recorded on Shure microphones. You can download the podcast direct, listen in-browser or subscribe through iTunes using the links below. Also, be sure to let us know your thoughts about the discussion in the forums.

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Source: http://feedproxy.google.com/~r/bit-tech/blog/~3/EUee9PuDJKs/

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Microsoft Japan prepares for Windows Phone Mango with new website

Inside the Mango update to the Windows Phone 7 platform is support for 17 new languages, one of which is Japanese.

In preparation for that upgrade, Microsoft Japan today launched a website to inform customers about Mango, and Windows Phone 7 in general. Other newly supported languages are: Brazilian Portuguese, Chinese (simplified and traditional), Czech, Danish, Dutch, Finnish, Greek, Hungarian, Korean, Norwegian (Bokmål), Polish, Portuguese, Russian, and Swedish.

You can visit the site here, or simply imbibe the following screenshot:

Mango will also include a new keyboard option to support typing in Japanese. A total of 20 new keyboards will be coming in Mango, to help diversify the potential userbase of the phone line.

Mango is set to debut no later than September, with many indicators pointing to an August release. At this time, there is no known formal release date. However, Microsoft appears to be making good on its promise to release the update before the Holiday season.

Given that the company seems set on making Mango available as quickly as possible, it could be that their strategy is to allow as much time as possible to both sell phones that run Mango before and through the holiday season, and also to provide time for carriers to test and roll out the update before the new year.

For now, Microsoft Japan is gearing up for the next several months. Microsoft depends on the global availability of Windows Phone 7 devices to bolster sales, a strategy that has yet to see real fruit. If Mango can turn that around is a question that will be answered by the end of 2011.

Update: According to friend of TNWmicrosoft, Tom Warren, this website is not quite as new as thought. As it turns out, it was released on VIP Mango day. The rest of the post is, of course, correct.

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Source: http://thenextweb.com/microsoft/2011/07/19/microsoft-japan-prepares-for-windows-phone-mango-with-new-website/

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